KIR Ligand Calculator Help
Recent transplant strategies based on KIR-ligand mismatch to predict NK cell alloreactivity have resulted in less relapse, less GvHD and better overall survival in patients with Acute Myeloid Leukaemia (AML) (1). KIR-ligands are HLA molecules that can be grouped into three major categories based on the amino acid sequence determining the KIR-binding epitope in HLA-C and HLA-B molecules. All expressed HLA-C alleles are of the C1 or C2 group (2) and most HLA-B alleles can be classified as either Bw4 or Bw6 (3). The grid below shows how the tool defines these groups. Killer-immunoglobin receptors KIR2DL1, KIR2DL2 and KIR3DL1 bind KIR-ligand C2, C1 and Bw4 respectively, resulting in inhibition of NK cell mediated lysis.
| Locus | Motif | 77 | 80 |
|---|---|---|---|
| HLA-B | Bw4 | N (Asparagine) | I (Isoleucine ) |
| HLA-B | Bw4 | N (Asparagine) | T (Threonine) |
| HLA-B | Bw4 | D (Aspartic acid) | T (Threonine) |
| HLA-B | Bw4 | S (Serine) | T (Threonine) |
| HLA-B | Bw6 | G (Glycine) | N (Asparagine) |
| HLA-B | Bw6 | S (Serine) | N (Asparagine) |
| HLA-C | C1 | N (Asparagine) | |
| HLA-C | C2 | K (Lysine) |
- Enter the two field typings in the box provided. For example for an individual typed as B*07:02, B*08:01, C*07:01, C*07:02, you would enter 07:02 in the first box, 08:01 in the second box and so on. The gene is already entered, so only the numerical part of the name needs adding.
- If an individual is homozygous for a locus enter the typing in all boxes. Do not leave a box blank.
- Null alleles are not expressed so please do not enter null alleles into the box provided.
- Two field typings are required.
Output
- The output lists the ligands associated with the typing entered.
- A link to the full list of alleles matching the type given and their associated ligands is provided.
References
All references are linked to the Medline abstract where possible.
- Role of natural killer cell alloreactivity in HLA-mismatched hematopoietic stem cell transplantation.Blood (1999) 91:333-9 (Abstract)
- HLA and NK cell inhibitory receptor genes in resolving hepatitis C virus infection.Science 305:872-4 (Abstract)
- Conserved and variable residues within the Bw4 motif of HLA-B make separable contributions to recognition by the NKB1 killer cell-inhibitory receptor.J Immunol. (1997) 158:5237-41 (Abstract)