Sample Instance

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{
    "data": {
        "type": "samples",
        "id": "SRS589380",
        "attributes": {
            "biosample": "SAMN02717005",
            "longitude": -113.23,
            "sample-metadata": [
                {
                    "key": "geographic location (longitude)",
                    "value": "-113.23",
                    "unit": null
                },
                {
                    "key": "geographic location (country and/or sea,region)",
                    "value": "Guangzhou",
                    "unit": null
                },
                {
                    "key": "environment (biome)",
                    "value": "microbiome",
                    "unit": null
                },
                {
                    "key": "geographic location (latitude)",
                    "value": "23.16",
                    "unit": null
                },
                {
                    "key": "instrument model",
                    "value": "454 GS FLX Titanium",
                    "unit": null
                },
                {
                    "key": "host scientific name",
                    "value": "Homo sapiens",
                    "unit": null
                }
            ],
            "latitude": 23.16,
            "accession": "SRS589380",
            "analysis-completed": "2018-01-24",
            "collection-date": null,
            "geo-loc-name": "Guangzhou",
            "sample-desc": "The strong correlation between bacterial communities and main factors (e.g., time and disease state) both between subjects and within subjects within a given niche thus suggested that oral microbiota could potentially model ECC onset. To test this hypothesis, the 34-children study cohort sampled two times during one year (T1, T3) were used as a train set for model construction, and an additional 24 child subjects were recruited and then each sampled at two time points (T3 and T4) for model validation: 14 patients including 7 experiencing caries-onset (HC group) and 7 experiencing caries- exacerbation (CC group) and ten healthy-steady controls (HH group).",
            "environment-biome": "microbiome",
            "environment-feature": null,
            "environment-material": null,
            "sample-name": "ECCK1V",
            "sample-alias": "ECCK1V",
            "host-tax-id": 9606,
            "species": "Homo sapiens",
            "last-update": "2018-01-24T09:37:01"
        },
        "relationships": {
            "biome": {
                "data": {
                    "type": "biomes",
                    "id": "root:Host-associated:Human:Digestive system:Oral"
                },
                "links": {
                    "related": "https://www.ebi.ac.uk/metagenomics/api/v1/biomes/root:Host-associated:Human:Digestive%20system:Oral?format=api"
                }
            },
            "studies": {
                "links": {
                    "related": "https://www.ebi.ac.uk/metagenomics/api/v1/samples/SRS589380/studies?format=api"
                },
                "data": [
                    {
                        "type": "studies",
                        "id": "MGYS00002171",
                        "links": {
                            "self": "https://www.ebi.ac.uk/metagenomics/api/v1/studies/MGYS00002171?format=api"
                        }
                    }
                ]
            },
            "runs": {
                "links": {
                    "related": "https://www.ebi.ac.uk/metagenomics/api/v1/samples/SRS589380/runs?format=api"
                }
            }
        },
        "links": {
            "self": "https://www.ebi.ac.uk/metagenomics/api/v1/samples/SRS589380?format=api"
        }
    }
}