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            "id": "MGYS00006499",
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                "accession": "MGYS00006499",
                "bioproject": "PRJNA419860",
                "samples-count": 194,
                "is-private": false,
                "last-update": "2023-09-25T13:53:45",
                "secondary-accession": "SRP125799",
                "centre-name": "University of Geneva",
                "public-release-date": null,
                "study-abstract": "Commensal microbiota play an important role in many physiological processes. Importantly, they pose a natural threshold to colonization with pathogenic bacteria by direct competition. Here we show that acute infection of the lung with influenza A viruses causes elimination of a substantial proportion of bacteria from the small intestine. This weakens the natural shield against pathogenic bacteria and renders the intestinal epithelium vulnerable for invading bacteria. We further show that influenza A virus infection up-regulates the activity of Paneth cells a major source of antimicrobial peptides in the small intestine. This suggests that influenza A viruses affect a host mechanism of microbiota regulation to cause bacterial depletion in the small intestine. Surprisingly, the same mechanism does not apply in the respiratory tract, where influenza A virus infection causes subtle qualitative but not quantitative changes in the commensal microbiota. Overall, our date provide the first description of microbiota dynamics in an acute infection animal model.",
                "study-name": "Lung microbiota of C57BL/6J mice (Mus Musculus) Raw sequence reads",
                "data-origination": "HARVESTED"
            },
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