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            "id": "MGYS00000533",
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                "accession": "MGYS00000533",
                "samples-count": 52,
                "bioproject": "PRJEB7112",
                "is-private": false,
                "last-update": "2016-01-20T14:12:06",
                "secondary-accession": "ERP006800",
                "centre-name": "WEIZMANN INSTITUE OF SCIENCE",
                "public-release-date": null,
                "study-abstract": "All domains of life feature diverse molecular clock machineries that synchronize physiological processes to diurnal environmental fluctuations. However, no mechanisms are known to cross-regulate prokaryotic and eukaryotic circadian rhythms in multi-kingdom ecosystems. Here, we show that the intestinal microbiota, in both mice and humans, exhibits diurnal oscillations that are influenced by feeding rhythms, leading to time-specific compositional and functional profiles over the course of a day. Ablation of host molecular clock components or induction of jet lag leads to aberrant microbiota diurnal fluctuations and dysbiosis, driven by impaired feeding rhythmicity. Consequently, jet lag-induced dysbiosis in both mice and humans promotes glucose intolerance and obesity that are transferrable to germ-free mice upon fecal transplantation. Together, these findings provide the first evidence of coordinated meta-organism diurnal rhythmicity, and offer a microbiome-dependent mechanism for common metabolic disturbances in humans with aberrant circadian rhythms, such as those documented in shift workers and frequent flyers.",
                "study-name": "Trans-kingdom control of microbiota diurnal oscillations promotes metabolic homeostasis",
                "data-origination": "SUBMITTED"
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