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E-GEOD-55645 - Genome wide analysis of copy number variation in NAFLD spectrum

Status
Released on 26 February 2015, last updated on 8 March 2015
Organism
Homo sapiens
Samples (98)
Array (1)
Protocols (5)
Description
Despite some success in identifying CNVs responsible for metabolic phenotypes including obesity and diabetes mellitus, there are as yet no data available to suggest whether or not CNVs might be involved in the etiology of the NAFLD spectrum. This report is a comprehensive analysis of copy number in Malaysian patients with NAFLD. Genomic DNA was extracted from blood obtained from patients with NAFLD and submitted for genome-wide analysis using aCGH
Experiment type
comparative genomic hybridization by array 
Contacts
Citation
Genome-wide analysis of copy number variation identifies candidate gene loci associated with the progression of non-alcoholic fatty liver disease. Zain SM, Mohamed R, Cooper DN, Razali R, Rampal S, Mahadeva S, Chan WK, Anwar A, Rosli NS, Mahfudz AS, Cheah PL, Basu RC, Mohamed Z. , PMID:24743702
MIAME
PlatformsProtocolsVariablesProcessedRaw
Files
Investigation descriptionE-GEOD-55645.idf.txt
Sample and data relationshipE-GEOD-55645.sdrf.txt
Raw data (25)Click to browse raw data
Processed data (1)E-GEOD-55645.processed.1.zip
Array designA-GEOD-9777.adf.txt
Links