InterPro

Literature: GTF2I-like repeat (IPR004212)

References used in this entry

• Identification of a putative transcription factor gene (WBSCR11) that is commonly deleted in Williams-Beuren syndrome.
  Osborne LR, Campbell T, Daradich A, Scherer SW, Tsui LC.
  Genomics 57 279-84 1999
  PMID: 10198167 Related citations
• Identification of a novel slow-muscle-fiber enhancer binding protein, MusTRD1.
  O'Mahoney JV, Guven KL, Lin J, Joya JE, Robinson CS, Wade RP, Hardeman EC.
  Mol. Cell. Biol. 18 6641-52 1998
  PMID: 9774679 Related citations

Further reading

• Comparison of TFII-I gene family members deleted in Williams-Beuren syndrome.
  Hinsley TA, Cunliffe P, Tipney HJ, Brass A, Tassabehji M.
  Protein Sci. 13 2588-99 2004
  PMID: 15388857 Related citations
• Multiple GTF2I-like repeats of general transcription factor 3 exhibit DNA binding properties. Evidence for a common origin as a sequence-specific DNA interaction module.
  Vullhorst D, Buonanno A.
  J. Biol. Chem. 280 31722-31 2005
  PMID: 15987678 Related citations
• Isolation and characterization of BEN, a member of the TFII-I family of DNA-binding proteins containing distinct helix-loop-helix domains.
  Bayarsaihan D, Ruddle FH.
  Proc. Natl. Acad. Sci. U.S.A. 97 7342-7 2000
  PMID: 10861001 Related citations
• Cloning of an inr- and E-box-binding protein, TFII-I, that interacts physically and functionally with USF1.
  Roy AL, Du H, Gregor PD, Novina CD, Martinez E, Roeder RG.
  EMBO J. 16 7091-104 1997
  PMID: 9384587 Related citations