Literature: GTF2I-like repeat (IPR004212)
References used in this entry
The following publications were referred to in the abstract:
Identification of a putative transcription factor gene (WBSCR11) that is commonly deleted in Williams-Beuren syndrome.
Osborne LR, Campbell T, Daradich A, Scherer SW, Tsui LC.
Genomics 57 279-84 1999
PMID: 10198167 Related citations
Identification of a novel slow-muscle-fiber enhancer binding protein, MusTRD1.
O'Mahoney JV, Guven KL, Lin J, Joya JE, Robinson CS, Wade RP, Hardeman EC.
Mol. Cell. Biol. 18 6641-52 1998
PMID: 9774679 Related citations
The following publications were not referred to in the abstract, but provide useful additional information:
Comparison of TFII-I gene family members deleted in Williams-Beuren syndrome.
Hinsley TA, Cunliffe P, Tipney HJ, Brass A, Tassabehji M.
Protein Sci. 13 2588-99 2004
PMID: 15388857 Related citations
Multiple GTF2I-like repeats of general transcription factor 3 exhibit DNA binding properties. Evidence for a common origin as a sequence-specific DNA interaction module.
Vullhorst D, Buonanno A.
J. Biol. Chem. 280 31722-31 2005
PMID: 15987678 Related citations
Isolation and characterization of BEN, a member of the TFII-I family of DNA-binding proteins containing distinct helix-loop-helix domains.
Bayarsaihan D, Ruddle FH.
Proc. Natl. Acad. Sci. U.S.A. 97 7342-7 2000
PMID: 10861001 Related citations
Cloning of an inr- and E-box-binding protein, TFII-I, that interacts physically and functionally with USF1.
Roy AL, Du H, Gregor PD, Novina CD, Martinez E, Roeder RG.
EMBO J. 16 7091-104 1997
PMID: 9384587 Related citations