Transcription profiling by array of cardic ventricle tissues from mice with cardiac-specific constitutive expression of an activated Akt (caAkt) and wild-type controls to test whether Akt activation contributes to mitochondrial dysfunction in pathological cardiac hypertrophy
Microarray 1-colour mRNA
Organism:
Mus musculus
Array Design(s):
Affymetrix GeneChip Mouse Genome 430 2.0 [Mouse430_2]
Publication:
- Wende AR, O'Neill BT, Bugger H, Riehle C, Tuinei J et al. (2015) Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.